effect on the QA-induced activation of

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Marions et al[15] reported that following mifepristone administration during early luteal phase in fertile women, down-regulation of PR was inhibited and no significant modifications of the remaining markers of endometrial receptivity, such as pinopodes, integrin dimmers a4 and b3, cyclo-oxygenase-1 and -2 were found.
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Synchronous expressions of fully developed pinopodes and other markers of uterine receptivity such as integrins (avb3)[16], heparin-binding epidermal growth factor (HB-EGF)[17], LIF and LIF-receptor[18] have been noted.
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